Today, I will discuss with everyone the diseases that induce cerebral thrombosis. These include a history of hypertension, abnormal ECG, heart disease, diabetes, hyperlipidemia, family history of hypertension, overweight, and preference for fatty meat, etc., which are related to the onset of cerebral thrombosis. The order is as follows: history of hypertension, systolic blood pressure, body mass index, and reduction in high-density lipoprotein, which are the main factors affecting the formation of cerebral thrombosis.
The formation of cerebral thrombosis is related to the following three factors:
(1) Vascular lesions: The most important and common vascular lesion is atherosclerosis, followed by cerebral small artery sclerosis associated with hypertension. Other causes include vascular developmental abnormalities such as congenital aneurysms and cerebrovascular malformations; vasculitis caused by infections like rheumatic fever, tuberculosis, leptospirosis, syphilis, etc., leading to arteritis; some non-infectious vasculitis such as thromboangiitis obliterans and nodular polyarteritis, arterial wall trauma from injuries, surgeries, catheters, or punctures; and rare cases of dissecting aneurysms in the aorta and carotid arteries.
(2) Changes in blood components: Rough endothelium at the site of vascular lesions makes platelets in the blood more prone to adhere, accumulate, and release more serotonin and other chemical substances. An increase in lipoprotein, cholesterol, and fibrinogen content in the blood can increase blood viscosity, slowing down blood flow. In addition, blood disorders such as leukemia, polycythemia, and various factors that increase blood coagulability also make it easier for cerebral thrombosis to form.
(3) Hemodynamic changes: The regulation of cerebral blood flow is influenced by many factors. Changes in blood pressure are important factors affecting local cerebral blood volume. When the mean arterial pressure is below 9.5 kPa (71 mmHg) and above 24 kPa (180 mmHg), due to pre-existing vascular lesions, narrowed lumens, and loss of autoregulatory function, the blood supply to local brain tissues may be impaired.
In addition, some researchers conducted a matched control study comparing patients with cerebral thrombosis with patients with other diseases. They collected over 30 components including age, gender, ethnicity, marital status, smoking, drinking, diet, mental stimulation, physical exercise, weight, blood pressure, blood sugar, ECG, serum cholesterol, etc., and performed regression analysis. They found that a history of hypertension, abnormal ECG, heart disease, diabetes, hyperlipidemia, family history of hypertension, overweight, and preference for fatty meat are related to the onset of cerebral thrombosis. The order is as follows: history of hypertension, systolic blood pressure, body mass index, and reduced high-density lipoprotein, which are the main factors affecting the formation of cerebral thrombosis. Physical exercise can reduce the occurrence of cerebral thrombosis.
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