Vulvar dysplasia is one of the difficult diseases in gynecology, and its pathogenesis mainly comes from three reasons: 1. Heredity or alternate generation heredity; 2. Lack or disorder of self-endocrine; 3. Decline of self-immune function. This disease is mainly divided into four types: hyperplastic type, simple atrophic type, lichen sclerosus type, mixed type, with stubborn and complex conditions. Mild cases present with increased leucorrhea, unbearable itching and pain; severe cases present with vulvar hyperplasia, sclerosis, atrophy, reduced sexual desire or no sexual desire. In particular, patients with simple atrophic type (no obvious itching or slight itching), if not treated in time, may cause urethritis, cystitis, nephritis, pyelonephritis, etc. Severe cases can lead to vulvar atrophy, loss of elasticity, complete disappearance of bilateral labia minora, adhesion of clitoris and prepuce, urethral atrophy, frequent urination, vaginal stenosis, urinary incontinence or urine shooting to both sides, continuous dripping, or urine flowing to the anus, painful intercourse accompanied by tears, or loss of sexual function. In the late stage, canceration is likely to occur.